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In earlier reported and recently updated studies, we've demonstrated that ALN-AAT may reduce liver degrees of mutant AAT, improve histopathology connected with mutant AAT expression, and reduce liver fibrosis and the incidence of tumor formation in a mouse model of alpha-1 liver disease, said Akshay Vaishnaw, M.D., Ph.D., Executive Vice President of R&D and Chief Medical Officer of Alnylam. The filing of the fresh CTA also highlights the reproducible and modular features of Alnylam's platform, as ALN-AAT at this point becomes our 6th clinical stage program inside our Genetic Medicine Superstar, our 7th clinical pipeline program general, and the fifth clinical program employing our ESC-GalNAc delivery technology.